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This approach enabled high-sensitivity quantification of dynamic changes in BCL-2 mRNA levels, AR expression, AR pathway activity ( KLK3, TMPRSS2 ), and potential compensatory or ‘bypass’ survival signaling (e.g., NR3C1 , i.e., GR) (Fig

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Through reducing glial cell inflammatory activity and enhancing glial viability, incretin-based multi-agonists may allow for reparative functions of glial cells to combat neuroinflammation and neurodegeneration