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In addition, the increase in proinflammatory cytokines leads to the formation of fibrin amyloid microclot resistant to fibrinolysis, either by increased production of serum amyloid A (SAA) or by the release of β-amyloid (Aβ) peptide by platelets, resulting in capillary obstruction (156–159)

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Other experimental forms of “immune therapy,” such as probiotics or helminth exposure designed to modulate immune responses, remain under investigation