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Because of the upregulation of mannose receptor (CD206) in fibrosis, delivery of siRNA targeting GTSE1 using mannose-modified lipid nanoparticles is an attractive strategy to reduce pro-EMT gene expression and improve lung function

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These cytokines contribute to the recruitment and activation of other immune cells, such as neutrophils, and promote the production of additional inflammatory mediators (Zong et al., 2025)

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